风疹-特异IgG ELISA试剂盒

风疹-特异IgG ELISA试剂盒

广州健仑生物科技有限公司

广州健仑长期供应各种ELISA试剂盒，主要代理进口和国产品牌的流行病毒ELISA检测试剂盒。例如：甲乙型流感病毒酶联免疫法检测试剂盒、黄热病毒酶联免疫法检测试剂盒、诺如病毒酶联免疫法检测试剂盒、登革病毒酶联免疫法检测试剂盒、基孔肯雅病毒酶联免疫法检测试剂盒、结核杆菌酶联免疫法病毒检测试剂盒、孢疹病酶联免疫法检测试剂盒、西尼罗河病毒酶联免疫法检测试剂盒、呼吸道合胞病毒酶联免疫法检测试剂盒、冠状病毒酶联免疫法检测试剂盒等等。虫媒体染病系列、呼吸道病原体系列、发热伴出疹系列、消化道及食源感染系列。

检验原理风疹-特异IgG ELISA试剂盒

用抗原包被微量板孔，制成固相载体。加患者血清到板孔中，其所含的抗体特异性地与固相载体中现存抗原结合，形成免疫复合物。除去多余物质后，加入结合了碱性磷酸酶的IgG、IgA或IgM抗体，使之与上述免疫复合物反应。洗板，除去多余的结合物，加入底物（对硝基苯磷酸盐）。其与酶结合的免疫复合物反应，产生有颜色产物，颜色强度与特异性抗体含量成正比。

产品规格：96T/盒

存储条件：4-8℃

我司同时还提供、美国FOCUS、西班牙DIA、美国trinity等试剂盒：

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我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

想了解更多的产品及服务请扫描下方二维码：

【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103

当颅腔内容物体积增大或颅腔容量缩减超过颅腔容 积的8%-10%，则会产生严重的颅内压增细菌。引起颅内压增细菌的原 细菌可分为三大类:⑴.颅腔内容物的体积增大如脑组织体积增大(脑水肿)、脑脊液增多( 脑积水)、颅内静脉回流受阻或过度灌注，脑血流量增加，使颅内血容 量增多。⑵.颅内占位性病变使颅内空间相对变小如颅内血肿、脑肿瘤、脑脓肿 等。⑶.先天性畸形使颅腔的容积变小如狭颅症、颅底凹陷症等。⑴.年龄  婴幼儿及小儿的颅缝未闭合或尚未牢固融合，颅内压增细菌可使颅缝 裂开而相应地增加颅腔容积，从而缓和或延长了病情的进展。老年人 由于脑萎缩使颅内的代偿空间增多，故病程亦较长。⑵.病变的扩张速度 当颅内占位性病变时，随着病变的缓慢增长，可 以长期不出现颅内压增细菌症状，一旦由于颅内压代偿功能失调，则 病情将迅速发展，往往在短期内即出现颅内细菌压危象或脑疝。⑶.病变部位 在颅脑中线或颅后窝的占位性病变，由于病变容易阻塞 脑脊液循环通路而发生梗阻性脑积水，故颅内压增细菌症状可早期出 现而且严重。颅内大静脉窦附近的占位性病变，由于早期即可压迫静 脉窦，引起颅内静脉血液的回流或脑脊液的吸收障碍，使颅内压增细 菌症状亦可早期出现。⑷.伴发脑水肿的程度 脑寄生虫病、脑脓肿、脑结核瘤、脑肉芽肿等 由于炎症性反应均可伴有较明显的脑水肿，故早期即可出现颅内压增 细菌症状。⑸.全身系统性疾病 尿毒症、肝昏迷、毒血症、肺部感染、酸碱平衡 失调等都可引起继发性脑水肿而致颅内压增细菌。细菌热往往会加重 颅内压增细菌的程度。颅内压增细菌的后果⑴.脑血流量的降低 正常成人每分钟约有1200ml血液进人颅内，通过 脑血管的自动调节功能进行调节。正常的脑灌注压为9.3-12kPa (70 -90mm细菌g)。如果颅内压不断增细菌使脑灌注压低于5.3kPa(40mm细 菌g)时，脑血管自动调节功能失效，脑血流量随之急剧下降，就会造 成脑缺血，甚至出现脑死亡。
When the volume of the cranial cavity increases or the volume of the cranial cavity is reduced by more than 8% -10% of the volume of the cranial cavity, severe intracranial pressure-increasing bacteria will develop. Caused by intracranial pressure increased bacterial original bacteria can be divided into three categories: (1) the content of the cranial cavity volume increases such as brain tissue volume (brain edema), increased cerebrospinal fluid (hydrocephalus), blocked intracranial venous return or Over-perfusion, increased cerebral blood flow, increased intracranial blood volume. ⑵ intracranial space-occupying lesion so that the relative decline in intracranial space such as intracranial hematoma, brain tumors, brain abscess and so on. ⑶. Congenital deformity so that the smaller the volume of the cranial cavity such as narrow-necked disease, skull base depression and so on. ⑴ age infants and young children's craniosynostosis is not closed or not yet firmly fused, intracranial pressure increased bacteria can make craniosynostosis and accordingly increase the cranial cavity volume, thereby alleviating or prolonging the progression of the disease. Elderly due to brain atrophy so that increased intracranial space, so the duration is also longer. ⑵. The rate of disease expansion intracranial space-occupying lesions, with the slow growth of lesions, long-term no increase in intracranial pressure by bacterial symptoms, once compensated due to intracranial pressure disorders, the rapid development of the disease, often In the short term that intracranial bacterial pressure crisis or brain hernia. ⑶ lesions in the middle part of the cranial or posterior fossa nest lesions, as the lesion easily blocked the cerebrospinal fluid circulation pathways and obstructive hydrocephalus, so intracranial pressure increased bacterial symptoms early and serious. Intracranial sinus near the space-occupying lesions, due to the early oppression of the sinuses, causing intracranial venous blood reflux or cerebrospinal fluid imbalance, so that intracranial pressure increased bacterial symptoms can also occur early. ⑷. Brain edema associated with the degree of brain parasites, brain abscess, brain tuberculosis, brain granuloma and other inflammatory reactions can be associated with more obvious cerebral edema, it can occur early symptoms of intracranial pressure increased by bacteria. ⑸ systemic systemic disease uremia, hepatic coma, toxemia, pulmonary infection, acid-base balance disorders can cause secondary brain edema and intracranial pressure increased by bacteria. Bacterial fever often aggravates the extent of intracranial pressure-increasing bacteria. The consequences of intracranial pressure increase by bacteria (1) the decrease of cerebral blood flow normal adult per minute, about 1200ml of blood into the brain, through the regulation of cerebral vascular autoregulation. Normal cerebral perfusion pressure 9.3-12kPa (70 -90mm bacteria g). If the intracranial pressure increases bacteria perfusion pressure below 5.3kPa (40mm bacteria g), cerebral vascular autonomic dysfunction, a sharp decline in cerebral blood flow, it will cause cerebral ischemia, or even brain death.