- 产品描述
嗜肺军团菌尿液检测试纸
广州健仑生物科技有限公司
(广州健仑生物科技有限公司是集研制开发、销售、服务于一体的优良企业,公司产品涉及临床快速诊断试剂、食品安全检测试剂,违禁品快速检测,动物疾病防疫检测试剂,免疫诊断试剂、临床血液学和体液学检验试剂、微生物检验试剂、分子生物学检验试剂、临床生化试剂、有机试剂等众多领域,同时核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名诊断产品集团公司产品,致力于为商检单位、疾病预防控制中心、海关出入境检疫局、卫生防疫单位,缉毒系统,戒毒中心,检验检疫单位、生化企业、科研院所、医疗机构等机构与行业提供*、高品质的产品服务。此外,本公司还开展食品、卫生、环境、药品等多方面的第三方检测服务。)
主要用途:用于检测尿样中嗜肺军团菌血清型1抗原,以支持军团菌感染的诊断。
产品规格:20T/盒
存储条件:2-30℃
嗜肺军团菌尿液检测试纸
我司还提供其它进口或国产试剂盒:登革热、疟疾、西尼罗河、立克次体、无形体、蜱虫、恙虫、利什曼原虫、RK39、汉坦病毒、深林脑炎、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。
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【产品介绍】
货号 | 产品名称 | 产品描述 | 产品规格 | 保存条件 |
JL-ET01 | 免疫捕获诺如病毒检测试剂盒 | 用于检测粪便标本中的诺如病毒抗原,以支持诺如病毒感染的诊断。 | 20T/盒 | 2-30℃ |
JL-ET02 | 免疫捕获军团菌检测试剂盒 | 用于检测尿样中嗜肺军团菌血清型1抗原,以支持军团菌感染的诊断。 | 20T/盒 | 2-30℃ |
JL-ET03 | 免疫捕获肺炎链球菌检测试剂盒 | 用于检测尿标本中的肺炎链球菌抗原,以支持肺炎链球菌感染的诊断。 | 20T/盒 | 2-30℃ |
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【公司名称】 广州健仑生物科技有限公司
【】 杨永汉
【】
【腾讯 】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化】
6.摄取与分泌神经递质
神经胶质细胞既能摄取,又能分泌神经递质。如:脑内星形胶质细胞能摄取谷氨酸与γ-氨基丁酸两种递质,以消除这两种递质对神经元的持续作用;同时又可通过星形胶质细胞的代谢,将这两种递质再转变为神经元可重新利用的递质前体物质。此外,星形胶质细胞还能合成并分泌血管紧张素原、前列腺素、白细胞介素以及多种神经营养病毒子等生物活性物质。神经胶质细胞通过对神经递质或生物活性物质的摄取、合成与分泌。而发挥其对神经元功能活动的调节作用。
7.吞噬作用
此外,小胶质细胞和星形胶质细胞有吞噬作用,能吞噬有病变的神经
1. 神经元急性坏死(红色神经元,red neuron):急性缺血、缺氧,急性中毒或感染可引起神经元的死亡,表现为神经元核固缩,胞体缩小变形,胞浆尼氏小体(Nissl body)消病毒,HE染色胞浆呈深伊红色,称为红色神经元(red neuron)。如细胞坏死后的酶性分解过程继续发展,则可导致细胞核溶解消病毒,残留细胞的轮廓或痕迹称为鬼影细胞(ghost cell)。由缺血引起的红色神经元zui常见于大脑皮质的锥体细胞和小脑蒲肯野(Purkinje)细胞。
2. 单纯性神经元萎缩(simple neuronal atrophy):神经元慢性渐进性变性以至死亡的过程称为单纯性神经元 萎缩。又称神经元的慢性病变。病变特点表现为神经元胞体缩小,核固缩而无明显的尼氏小体溶解,一般 不伴炎症反应。晚期可伴明显胶质细胞增生。
3. 中央性Nissl小体溶解(ce病毒al chromatolysis):为一种可逆性变性,常由病毒感染,维生素B缺乏及神经 元与轴突断离等病毒素所致。病变表现为神经元肿胀、变圆、核偏位,胞浆中央的尼氏小体崩解,进而溶解 消病毒,或仅在细胞周边部有少量残余,胞浆着色浅而呈苍白均质状。
4. 神经元胞质内包含体形成(i病毒ancyto-plasmic inclusion):神经元胞质内包含体形成可见于某些病毒感染 和变性疾病等,其形态、大小和着色不同,分布部位也有一定规律,如Parkinson病的黑质,蓝斑等处的神 经细胞中的Lewy小体(图14-2);狂犬病时海马和脑皮质锥体细胞中的Negri小体,分别对这些疾病具有诊 断意义。此外,神经元胞浆中出现脂褐素多见于老年人。
6. Uptake and secretion of neurotransmitters
Glial cells can not only uptake, but also neurotransmitters. Such as: astrocytes in the brain can take up glutamate and γ-aminobutyric acid two neurotransmitters in order to eliminate the two neurotransmitters on the sustained effect; at the same time through the astrocyte metabolism , Converting these two neurotransmitters into neurotransmitter precursors. In addition, astrocytes also synthesize and secrete biologically active substances such as angiotensinogen, prostaglandins, interleukins and various neurotrophic viruses. Glial cells through the neurotransmitter or bioactive substances uptake, synthesis and secretion. And play its role in the regulation of neuronal functional activity.
7. phagocytosis
In addition, microglia and astrocytes have phagocytosis that engulfs diseased nerves
1. Neuronal Acute Necrosis (red neuron): Acute ischemia, hypoxia, acute poisoning or infection can cause neuronal death, manifested as neuron nuclear condensation, cell body deformation, cytoplasmic Nissl small Body (Nissl body) virus, HE staining cytoplasm dark red, known as red neurons (red neuron). If the process of enzymatic decomposition after necrosis continues, the nuclei will dissolve the virus and the outline or trace of residual cells will be called the ghost cell. Red neurons caused by ischemia are most commonly found in the pyramidal cells of the cerebral cortex and in Purkinje cells of the cerebellum.
2. Simple atrophy of nerve cells (simple neuronal atrophy): The process of chronic progressive degeneration and death of neurons is called simple neuronal atrophy. Also known as neuronal chronic disease. Lesions characterized by shrinkage of neuronal somatic cells, nuclear pyknosis and no significant Nissl body lysis, generally do not have inflammatory reactions. Late with obvious glial cell proliferation.
3. Central Nissl body lysis (ce virus al chromatolysis): a reversible degeneration, often caused by viral infection, vitamin B deficiency and neurons and axons off the other toxins. Lesions showed neuronal swelling, rounding, nuclear deviation, the central cytoplasmic Nissl body disintegration, and then dissolve the virus, or only a small amount of residual cells in the periphery, the pale cytoplasm was pale homogeneous shape .
4. Neuronal cytoplasmic inclusion body formation (i virus ancyto-plasmic inclusion): Neuronal cytoplasmic inclusion bodies can be seen in some viral infections and degenerative diseases, the shape, size and coloring of different distribution sites also have a Such as Lewy bodies in neurons at the substantia nigra and locus coeruleus of Parkinson's disease (Figure 14-2); Negri bodies in the hippocampus and pyramidal cells of rabbits at the time of rabies are diagnostic for these diseases, respectively . In addition, the presence of lipofuscin in neuronal cytoplasm is more common in the elderly.