编号	英文名称	中文名称
JL-FA-01	Amebiasis (AME)	阿米巴病
JL-FA-02	Allergens, Rast scores	过敏原，放射性过敏原吸收实验。指对特定的人群引起免疫反应或者过敏反应的食品中的蛋白质
JL-FA-03	Allergens, Rast scores negative	过敏原，放射性过敏原吸收实验阴性
JL-FA-04	Anti-cyclic citrullinated peptide Antibody (CCP) Arthritis	抗环瓜氨酸肽抗体
JL-FA-05	ASCA Saccharomyces Cerevi	人抗酿酒酵母抗体（ASCA）
JL-FA-06	Aspergillis	麴菌病
JL-FA-07	Beta 2 Glycoprotein	β2糖蛋白
JL-FA-08	Beta 2 Glycoprotein IgM	β2糖蛋白 IGM
JL-FA-09	Bordela Pertussis	百日咳杆菌
JL-FA-10	Bordela Pertussis IgM	百日咳杆菌 IGM
JL-FA-11	C-ANCA	C-抗中性粒细胞胞浆抗体（ANCA)
JL-FA-12	Cardiolipin	心肌磷脂
JL-FA-13	Cardiolipin IgA	心肌磷脂 IGA
JL-FA-14	Cardiolipin IgG	心肌磷脂 IGG
JL-FA-15	Cardiolipin IgM	心肌磷脂 IGM
JL-FA-16	Cerebral Spinal Fluid	脑脊髓液
JL-FA-17	Chagas	恰加斯病/南美锥虫
JL-FA-18	Chlamydia	衣原体
JL-FA-19	Chlamydia IgA	衣原体IGA
JL-FA-20	Chlamydia IgG	衣原体IGG
JL-FA-21	Chlamydia IgM	衣原体IGM
JL-FA-22	Chlamydia Neg	衣原体阴性
JL-FA-23	Clotting Factor C3	凝固因子C3
JL-FA-24	Clotting Factor C4	凝固因子C4
JL-FA-25	Coccidiodes	球孢菌
JL-FA-26	Cytomegalovirus (CMV) Neg	巨细胞病毒抗体阴性
JL-FA-27	CMV IgG	巨细胞病毒 IGG阳性
JL-FA-28	CMV IgM VCA	巨细胞病毒 IGM 阳性
JL-FA-29	C-Reactive Protein (CRP)	C-反应蛋白质
JL-FA-30	Dengue Fever	登革热
JL-FA-31	Dengue Fever IgM	登革热 IGM
JL-FA-32	DS (Double Stranded) DNA	双链脱氧核糖核酸
JL-FA-33	EBNA (Epstein-Barr nuclear antigen) IgG	EB病毒核抗原 IGG
JL-FA-34	EBNA (Epstein-Barr nuclear antigen) IgM	EB病毒核抗原 IGM
JL-FA-35	Epstein Barr Virus (EBV) Negative Plasma	EB病毒阴性血浆
JL-FA-36	Epstein Barr Virus (EBV) EA IgM	EB病毒早期抗原 IGM
JL-FA-37	Epstein Barr Virus (EBV) VCA IgM	EB病毒壳蛋白 IGM
JL-FA-38	Epstein Barr Virus (EBV) EA IgG	EB病毒早期抗原 IGG
JL-FA-39	EMA (Endomysial Antibodies)	肌内膜
JL-FA-40	Gliadin	麸蛋白,麦醇溶蛋白,麦胶蛋白
JL-FA-41	Gliadin IgG	麦醇溶蛋白 IGG
JL-FA-42	Gliadin IgA	麦醇溶蛋白 IGA
JL-FA-43	Glomerular Basement Membrane (GBMA)	肾小球基底膜病
JL-FA-44	Helicobacter pylori IgA	幽门螺旋杆菌IGA
JL-FA-45	Helicobacter pylori IgG	幽门螺旋杆菌IGG
JL-FA-46	Helicobacter pylori IgM	幽门螺旋杆菌IGM
JL-FA-47	Helicobacter pylori Negative	幽门螺旋杆菌阴性
JL-FA-48	Helicobacter pylori Positive Plasma	幽门螺旋杆菌阴性血浆
JL-FA-49	Hepatitis A Virus (HAV) Pos. Plasma	甲型肝炎病毒阳性血浆
JL-FA-50	Hepatitis A Virus (HAV) IgM	甲型肝炎病毒IGM
JL-FA-51	Hepatitis B Core (HBc) IgG	乙型肝炎病毒核心 IGG
JL-FA-52	Hepatitis B Core (HBc) IgM	乙型肝炎病毒核心 IGM
JL-FA-53	Anti Hbe (Antibody to HBV antigen)	乙肝抗体
JL-FA-54	Hepatitis Delta Virus	丁型肝炎病毒
JL-FA-55	HBeAg (HBV e antigen)	乙肝 E抗原
JL-FA-56	anti-HBs (HBV surface antibody)	乙肝表面抗体
JL-FA-57	Hepatitis B (HBsAg) "Chronic"	乙型肝炎（乙肝表面抗原）“慢性病
JL-FA-58	HBsAg (HBV surface antigen) Serum	乙肝表面抗原血清
JL-FA-59	HBsAg (AD)	乙肝表面抗原（AD）
JL-FA-60	HBsAg (AY)	乙肝表面抗原（AY）
JL-FA-61	HBV Positive Plasma	乙肝阳性血浆
JL-FA-62	HBV DNA Plasma	乙肝DNA血浆
JL-FA-63	HBV DNA Serum	乙肝DNA血清
JL-FA-64	HBV DNA type A	A型乙肝DNA
JL-FA-65	HBV DNA type B	B型乙肝DNA
JL-FA-66	HBV DNA type C	C型乙肝DNA
JL-FA-67	HBV DNA type D	D型乙肝DNA
JL-FA-68	HBV DNA type E	E型乙肝DNA
JL-FA-69	HBV DNA type F	F型乙肝DNA
JL-FA-70	HBV Antibody HCV Antibody Plasma CO-INFECTED	乙肝和丙肝联合感染血浆
JL-FA-71	HCV (Hepatitis C Virus) Antibody	丙型肝炎抗体
JL-FA-72	HCV Core Antigen Positive	丙肝核心抗原阳性
JL-FA-73	HCV RNA PLASMA Genotype 1	基因1型丙肝RNA 血浆
JL-FA-74	HCV RNA PLASMA Genotype 2	基因2型丙肝RNA 血浆
JL-FA-75	HCV RNA PLASMA Genotype 3	基因3型丙肝RNA 血浆
JL-FA-76	HCV RNA PLASMA Genotype 4	基因4型丙肝RNA 血浆
JL-FA-77	HCV RNA PLASMA Genotype 5	基因5型丙肝RNA 血浆
JL-FA-78	HCV RNA PLASMA Genotype 6	基因6型丙肝RNA 血浆
JL-FA-79	HCV Riba single band	丙肝免疫印迹单波段
JL-FA-80	HCV RIBA Pos. (multiple bands)	丙肝免疫印迹阳性多波段
JL-FA-81	HCV Negative	丙肝阴性
JL-FA-82	HCV RNA Pos (quantitative)	丙肝RNA阳性（定量）
JL-FA-83	Hepatitis E	戊型肝炎
JL-FA-84	Herpes Simplex Virus (HSV)1/2 Positive Plasma	单纯性疱疹病毒1/2阳性血浆
JL-FA-85	Herpes Simplex Virus (HSV) 1 Negative Plasma	单纯性疱疹病毒1 阴性血浆
JL-FA-86	Herpes Simplex Virus (HSV) 1 IgG	单纯性疱疹病毒1 IGG
JL-FA-87	Herpes Simplex Virus (HSV 1) IgM	单纯性疱疹病毒1 IGM
JL-FA-88	Herpes Simplex Virus (HSV) 2 IgG	单纯性疱疹病毒2 IGG
JL-FA-89	Herpes Simplex Virus (HSV) 2 IgM	单纯性疱疹病毒2 IGG
JL-FA-90	Histone	组蛋白
JL-FA-91	Human Anti Mouse Ab (HAMA)	人抗鼠抗体
JL-FA-92	Human immunodeficiency virus (HIV) 1 Neg	HIV I 阴性
JL-FA-93	anti Human immunodeficiency virus (HIV) 1 Plasma	抗HIV I 血浆
JL-FA-94	anti Human immunodeficiency virus (HIV) 1 Serum	抗HIV I 血清
JL-FA-95	anti Human immunodeficiency virus (HIV) 2 Western Blot Tested	抗HIV 2 免疫印迹
JL-FA-96	anti Human immunodeficiency virus (HIV) 1/2 2 HIV (+)	抗HIV 1/2 2 HIV阳性
JL-FA-97	Human immunodeficiency virus (HIV) Ag	HIV抗原
JL-FA-98	HIV RNA (quantitative) Plasma	HIV RNA 定量血浆
JL-FA-99	HIV RNA (quantitative) Serum	HIV RNA 定量血清
JL-FA-100	HIV1 Subtype A	HIV1 亚型A
JL-FA-101	HIV1 Subtype B	HIV1 亚型B
JL-FA-102	HIV1 Subtype C	HIV1 亚型C
JL-FA-103	HIV1 Subtype D	HIV1 亚型D
JL-FA-104	HIV1 Subtype E	HIV1 亚型E
JL-FA-105	HIV1 Subtype F	HIV1 亚型F
JL-FA-106	HIV1 Subtype G	HIV1 亚型G
JL-FA-107	HIV1 Subtype H	HIV1 亚型H
JL-FA-108	HIV1 Subtype J	HIV1 亚型J
JL-FA-109	HIV1 Subtype K	HIV1 亚型K
JL-FA-110	HIV1 Group O	HIV1 亚型O
JL-FA-111	Human immunodeficiency virus (HIV) 2 Antibody Plasma	HIV 2 抗体血浆
JL-FA-112	Human immunodeficiency virus (HIV) 2 Antibody Serum	HIV 2 抗体血清
JL-FA-113	HPV (Human Papiloma Virus) Negative	人乳状瘤病毒HPV阴性
JL-FA-114	HPV (Human Papiloma Virus) Positive	人乳状瘤病毒HPV阳性
JL-FA-115	Human immunodeficiency virus (HIV) Antibody HCV Antibody Plasma COINFECTED	HIV 抗体 HCV
JL-FA-116	Human T-cell Lymphotropic Virus (HTLV) I/II	人嗜T淋巴细胞病毒(HTLV) I/II
JL-FA-117	Human T-cell Lymphotropic Virus (HTLV) I	人嗜T淋巴细胞病毒(HTLV) I
JL-FA-118	Human T-cell Lymphotropic Virus (HTLV) II	人嗜T淋巴细胞病毒(HTLV) II
JL-FA-119	Jo-1	多发性肌炎抗原JO-1
JL-FA-120	IgE < 5,000 Ku/L	IgE < 5,000 Ku/L
JL-FA-121	Legionella	军团杆菌属
JL-FA-122	Leptospira	军团杆菌属
JL-FA-123	Lyme Disease	莱姆(氏)病:蜱传播的全身性疾病,常在夏季发生
JL-FA-124	Lyme IgG	莱姆(氏)病 IGG
JL-FA-125	Lyme IgM	莱姆(氏)病 IGM
JL-FA-126	Lyme Disease Neg	莱姆(氏)病阴性
JL-FA-127	Malaria	疟疾
JL-FA-128	Mononucleosis (infectious)	单核细胞增多症（有传染性的）
JL-FA-129	Mononucleosis Negative	单核细胞增多症阴性
JL-FA-130	Measles Negative	麻疹阴性
JL-FA-131	Measles IgG	麻疹 IGG
JL-FA-132	Measles IgM	麻疹 IGM
JL-FA-133	Microsomal Anti-thyroid peroxidase antibody (TPO) Positive Plasma Standard Titer (typically 1,000-3,000 IU/mL)	微粒体抗甲状腺过氧化物酶抗体
JL-FA-134	Microsomal Anti-thyroid peroxidase antibody (TPO) Negative Plasma	微粒体抗甲状腺过氧化物酶抗体
JL-FA-135	Anti-mitochondrial antibody (AMA)	抗线粒体抗体
JL-FA-136	Multiple Sclerosis	多发性硬化症
JL-FA-137	Mumps IgG	流行性腮腺炎 IGG
JL-FA-138	Mumps Ab IgM	流行性腮腺炎抗体 IGM
JL-FA-139	Mumps Antibody Negative Plasma	流行性腮腺炎抗体阴性血浆
JL-FA-140	Mumps Antibody Negative Serum	流行性腮腺炎抗体阴性血清
JL-FA-141	Myeloma Plasma	骨髓瘤血浆
JL-FA-142	Myeloma IgA	骨髓瘤IGA
JL-FA-143	Myeloma IgE	骨髓瘤IGE
JL-FA-144	Myeloma IgG	骨髓瘤IGG
JL-FA-145	Myeloma IgM	骨髓瘤IGM
JL-FA-146	Mycoplasma	支原体
JL-FA-147	Mycoplasma Negative	支原体阴性
JL-FA-148	Mycoplasma IgG	支原体IGG
JL-FA-149	Mycoplasma IgM	支原体IGM
JL-FA-150	Mycoplasma PCR	支原体PCR
JL-FA-151	Normal Human Plasma	正常人血浆
JL-FA-152	Normal Human Serum	正常人血清
JL-FA-153	Nuclear Antibody Centromere	核抗体着丝粒
JL-FA-154	Nuclear Antibody, Speckled ANA	核抗体，斑点抗核抗体
JL-FA-155	Nuclear Antibody, Nucleolar ANA	核抗体,核仁抗核抗体
JL-FA-156	Nuclear Antibody, Homogeneous ANA	核抗体，同质抗核抗体
JL-FA-157	Nuclear Antiobody, Speckled. (ANA) Negative	核抗体，斑点。抗核抗体阴性
JL-FA-158	P-ANCA (associated neutrophil cytoplasmic antibodies)	相关的嗜中性粒细胞胞浆抗体
JL-FA-159	Parietal Cell Antibody (PCA)	胃)壁细胞抗体
JL-FA-160	Parvo positive plasma	细小病毒阳性血浆
JL-FA-161	Parvo IgM	细小病毒 IGM
JL-FA-162	Parvo IgG	细小病毒 IGG
JL-FA-163	Parvo Negative Plasma	细小病毒阴性血浆
JL-FA-164	Parvo DNA positive	细小病毒 DNA 阳性
JL-FA-165	Phospholipid Positive Plasma	磷脂阳性血浆
JL-FA-166	Prothrombin	凝血酶原,凝血因子
JL-FA-167	Rheumatoid Factor (RF) <1000 IU/mL	类风湿因子<1000 IU/mL
JL-FA-168	Rheumatoid Factor (RF) 1001-2000 IU/mL	类风湿因子1001-2000 IU/mL
JL-FA-169	Rheumatoid Factor (RF) 2001-4000 IU/mL	类风湿因子 2001-4000 IU/mL
JL-FA-170	Rheumatoid Factor (RF) 4001-5000 IU/mL	类风湿因子 4001-5000 IU/mL
JL-FA-171	Rheumatoid Factor (RF) >5000 IU/mL	类风湿因子>5000 IU/mL
JL-FA-172	Ribonucleoprotein (RNP) Positive	核糖核蛋白阳性
JL-FA-173	Rubella Chimeric	风疹
JL-FA-174	Rubella Negative	风疹阴性
JL-FA-175	Rubella IgG	风疹IGG
JL-FA-176	Rubella IgM	风疹IGM
JL-FA-177	Rubeola Negative Plasma	风疹阴性血浆
JL-FA-178	Rubeola IgG	风疹IGG
JL-FA-179	Scleroderma (Scl-70) Pos	胶原沉着病,硬皮病,硬皮症阳性
JL-FA-180	Scleroderma (Scl-70) Negative	硬皮病阴性
JL-FA-181	Sickle Cell Fresh Whole Blood	镰刀形红细胞新鲜全血
JL-FA-182	Smith (SM)	抗Smith抗体阳性血清（SLE的特征性抗体）
JL-FA-183	SMITH RNP	抗RNP抗体阳性血清（SLE的特征性抗体）
JL-FA-184	Smooth Muscle (ASMA)	抗平滑肌抗体阳性血清
JL-FA-185	Sjogren syndrome antigen A (SSA) Positive	舍格伦综合征或干燥综合征抗原A 阳性
JL-FA-186	Sjogren syndrome antigen B (SSB) Positive	舍格伦综合征抗原B 阳性
JL-FA-187	Sjogren syndrome antigen B (SSB) Negative	舍格伦综合征抗原B阴性
JL-FA-188	Streptolysin O Ab (ASO)	链球菌溶血素O抗体
JL-FA-189	Syphilis (RPR - Rapid Plasma Reagin) Positive Plasma	梅毒（梅毒-快速血浆反应）阳性血浆
JL-FA-190	Syphilis (RPR - Rapid Plasma Reagin) Negative Plasma	梅毒（梅毒-快速血浆反应）阴性血浆
JL-FA-191	Syphilis/ATA/T. pallidum IgG	梅毒ATA/T，苍白球IGG
JL-FA-192	Syphilis/ATA/T. pallidum IgM	梅毒ATA/T，苍白球IGM
JL-FA-193	Systemic Lupus Erythematosus (SLE) Positive	全身性红斑狼疮阳性
JL-FA-194	Systemic Lupus Erythematosus (SLE) Negative	全身性红斑狼疮阴性
JL-FA-195	TG/TPO Positive (Standard Titer 1,000 - 3000 IU/mL)	甲状腺球蛋白/甲状腺过氧化物酶阳性
JL-FA-196	TG/TPO Negative	甲状腺球蛋白/甲状腺过氧化物酶阴性
JL-FA-197	TTG (Tissue Transglutaminase)	组织转谷氨酰胺酶
JL-FA-198	TTG (Tissue Transglutaminase) IgA	组织转谷氨酰胺酶 IGA
JL-FA-199	ToRCH (Toxo, Rubella, CMV, HSV) Positive	优生优育（弓形虫，风疹，巨细胞，单胞）阳性
JL-FA-200	ToRCH (Toxo, Rubella, CMV, HSV) Negative	优生优育（弓形虫，风疹，巨细胞，单胞）阴性
JL-FA-201	Toxoplasmosis (Toxo)	弓形虫病
JL-FA-202	Toxoplasmosis (Toxo) IgG	弓形虫病IGG
JL-FA-203	Toxoplasmosis (Toxo) IgM	弓形虫病IGM
JL-FA-204	Thyroglobulin (TG) Positive Plasma	甲状腺球蛋白阳性血浆
JL-FA-205	Thyroglobulin (TG) Negative	甲状腺球蛋白阴性
JL-FA-206	Varicella-Zoster Virus (VZV) Negative	水痘-带状疱疹病毒阴性
JL-FA-207	Varicella-Zoster Virus (VZV) IgG	水痘-带状疱疹病毒IGG
JL-FA-208	Varicella-Zoster Virus (VZV) IgM	水痘-带状疱疹病毒IGM
JL-FA-209	West Nile Virus (WNV)	西尼罗河脑炎病毒
JL-FA-210	West Nile Virus (WNV) IgM	西尼罗河脑炎病毒IGM

美国

在小鼠中进行的这项研究也显示肾脏自我更新的能力。不是单一型的肾干细胞就可替换任何丢失或受损的肾脏组织，而是驻留在肾脏的不同部分稍微??更专门的干细胞产生每个类型的肾组织??内的新细胞。
“这就像有分支的树，其中每个分支考虑其自身的成长，而不是依赖于躯干的照顾，”德克尔说。
科学家还发现，这些细胞是通过一个名为Wnt蛋白的细胞通路的活化作出生长的决定。Rinkevich说，即使肾上皮细胞的数量看起来一样，zui健壮肾脏形成能力可追溯到前体细胞中的Wnt信号被激活，并且只能生长为某些特定类型的肾组织。“这提示了Wnt信号是负责新肾组织的成长提供了一个治疗目标，以通过促进或恢复肾脏的再生能力，”他说。“我们也许能够打开Wnt信号通路产生新的肾脏形成细胞。”
研究人员说，这项研究发现对于科学家试图在实验室创造肾部分有着至关重要的影响。
然而，他们警告说，这样的进步并不迫在眉睫。“为了在实验室里长出整个肾脏是复杂的，因为我们需要协调许多不同种类的前体细胞的活性，” 德克尔说。“这不象血液和免疫系统可以从一种类型的干细胞再生。”
奥地利科学技术研究所(IST)的副教授Harald Janovjak，与维也纳医学大学癌症研究学会的副教授迈克尔·格吕施，一起利用光“遥控”癌细胞的行为，相关文章发表于本周的EMBO杂志上。这项工作*将光遗传学应用到癌症研究的新领域。
为了解细胞信号的动态，研究人员需要将膜受体蛋白激活和使其失活，膜受体蛋白作为一个细胞内外世界之间的中转站。理想情况下，这种激活在短时间(几秒到几分钟)以及近的目标位置(微米到毫米)内发生。然而，如此高水平的活化精密度不能使用当前的药理学和遗传学的方法来实现。光遗传学利用光来控制细胞的活性，以及具有在时间和空间上精确地被应用和移动(施加和除去)的优点。Janovjak，格吕施和同事重新设计受体酪氨酸激酶(RTKs)，在光的控制下，感测生长因子和激素的必要的细胞表面受体。
当一个信号分子结合到细胞表面的RTKs，两种受体相互结合的过程称为二聚化作用。这个过程会激活细胞的信号转导。Janovjak，格吕施和同事将哺乳动物的RTKs的那些部分与光 - 氧 - 电压传感领域(鉴定黄绿藻的可逆光传感器)结合。在工程化受体中，二聚化步骤和随后的细胞信号转导现在可以通过光随着藻蛋白光感打开和关闭，以及彼此结合。在肿瘤细胞中，工程受体的活化导致细胞形态变化，细胞增殖和基因表达，增加癌症的恶性程度的特性。在血细胞中，活化导致细胞出芽，典型的新血管形成。

美国

二维码扫一扫

【公司名称】 广州健仑生物科技有限公司
【】杨永汉
【】
【腾讯】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化】

This study, conducted in mice, also shows the kidney's ability to self-renew. Rather than a single type of kidney stem cell that replaces any lost or damaged kidney tissue, it resides in a slightly more specialized, more specialized stem cell that produces new cells within each type of kidney tissue.
"It's like a tree with branches, each of which takes into account its own growth, not its torso care," Decker said.
Scientists also found that these cells are the decision to grow through the activation of a cellular pathway called Wnt protein. Rinkevich said that even though the number of renal epithelial cells looks the same, the most robust kidney formation can be traced back to the activation of Wnt signaling in precursor cells and can only grow to certain types of kidney tissue. "This suggests that Wnt signaling is responsible for the growth of new kidney tissue and provides a therapeutic target to promote or restore the regenerative capacity of the kidneys," he said. "We may be able to turn on the Wnt signaling pathway to produce new kidney-forming cells."
The researchers said the study found a crucial impact on scientists trying to create parts of the kidney in the lab.
However, they warned that such progress is not imminent. "To grow an entire kidney in a lab is complicated because we need to coordinate the activity of many different kinds of precursor cells," Decker said. "It's not like the blood and the immune system can regenerate from one type of stem cell."
Harald Janovjak, an associate professor at the Austrian Institute of Science and Technology (IST), and Michael Gruissch, associate professor at the Society for Cancer Research at the Medical University of Vienna, use light to "control" cancer cells in an article published this week in the EMBO journal. This work, for the first time, applies optogenetics to new areas of cancer research.
To understand the dynamics of cell signaling, researchers need to activate and inactivate membrane receptor proteins, which act as a transit site between the cell's inner and outer cells. Ideally, this activation occurs within short periods of time (seconds to minutes) and near target locations (micrometers to millimeters). However, such a high level of activation precision can not be achieved using current pharmacological and genetic approaches. Photogenetics uses light to control the activity of cells, and has the advantage of being precisely applied and moved (applied and removed) in time and space. Janovjak, Gruch and colleagues redesigned their receptor tyrosine kinases (RTKs) to sense the necessary cell surface receptors for growth factors and hormones under the control of light.
When a signaling molecule binds to a cell surface RTKs, the two receptors bind to each other as a dimerization. This process activates cell signaling. Janovjak, Gruch and colleagues combined those parts of the mammalian RTKs with the field of photo-oxygen-voltage sensing, a reversible light sensor that identifies Chlorella. In engineered receptors, the dimerization step and subsequent cellular signal transduction can now be turned on and off by light along with the algin protein and bind to each other. In tumor cells, activation of engineered receptors leads to changes in cell morphology, cell proliferation and gene expression, and increases the malignancy of cancers. In blood cells, activation leads to cell budding, typical neovascularization.

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